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Importantly, HO-1 can be targeted pharmacologically by interventions that seek its upregulation or enhancement of its various arms of action, via: (1) induction of HO-1 expression with drugs such as heme arginate, (2) administration of CO by inhalation or use of CO releasing molecules (CORM), and (3) administration of biliverdin/bilirubin or use of inhibitors of bilirubin conjugation that would result in increased bilirubin levels

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used a network pharmacology approach to predict that Catalpol could inhibit ferroptosis and mitigate fluoxetine-induced liver injury

M., Keasler J

89 Results from several studies point to a correlation between the NF1 and oestrogen receptor status in multiple model systems

Blood 92, 18701877