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Taken together with our metabolomic profiling of L-cystine starved cells (Fig

Setting aside the economic impact of IBD, the individual suffering brought about by these diseases is immeasurable

Notably, HIF-1 mediates adaptive metabolic responses to hypoxia by enhancing glycolytic pathway, serine synthesis and one-carbon metabolism to promote mitochondrial antioxidant production (NADPH and GSH), and inhibiting the TCA cycle so as to diminish mitochondrial ROS production (247)

However, it doesn't address the underlying immune dysregulation

Therefore, the formation and accumulation of OH leads to cell death [45]

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