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Through its modulation of the NNMT-SAM-NAD+ axis, it enhances energy expenditure and supports more efficient utilization of fat stores for energy, making it a strong candidate in studies exploring metabolic optimization and obesity control

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The increase in PrP levels appears to allow excess Cu to bypass defense mechanisms activated by ATP7B-KO cells to limit Cu uptake, such as the downregulation of the high-affinity copper transporter CTR1 13,39

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