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Phosphorylated -catenin is recognized by the -TrCP and subsequently degraded via the ubiquitin-dependent proteasome pathway

Due to the special characteristics of the membrane components, it promotes the bending and flow of the membrane

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Surgical treatment of pulmonary complications in Behets syndrome

Chronic 3-NP administration depletes ATP and NAD + levels and increases the release of ILs, TNF-, glutamate, and iNOS [168, 169]