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Traditional treatment approaches focus primarily on reducing stomach acid production through proton pump inhibitors or H2 blockers

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The catalytic tetrad formed by Sec, glutamine, tryptophan and asparagine is essential for GPx activity, since these residues are crucial for enzyme-substrate interaction and stabilization of the GSH-GPx interaction [47, 65, 66]

Each vial pairs three signaling molecules studied for distinct repair steps

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Neuroinflammation also emerges as a critical determinant: elevated cytokines such as TNF- , IL-1, and IL-6 destabilize synaptic homeostasis, impair inhibitory neurotransmission within descending pain pathways, and exacerbate central sensitization, ultimately limiting the therapeutic impact of tDCS (9698)