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Top panels illustrate mechanisms of action: atropine (2.1 mg) competitively antagonizes acetylcholine at muscarinic receptors to reverse cholinergic effects (e.g., bronchorrhea, bronchospasm, bradycardia), while pralidoxime (2PAM, 600 mg) reactivates acetylcholinesterase by cleaving the nerve agentenzyme bond prior to irreversible aging. Middle panels depict the dualchamber autoinjector design, which contains atropine and pralidoxime in separate compartments and delivers both drugs sequentially via a springloaded intramuscular mechanism from a single needle

Beutler E, Felitti VJ, Koziol JA, Ho NJ, Gelbart T

Always consult your own health-care provider for medical guidance.

Glucose-6-phosphate dehydrogenase deficiency in the Han Chinese population: molecular characterization and genotype-phenotype association throughout an activity distribution

GSTP1*B is an allele in which a single nucleotide (AG) substitution at position 313 substantially diminishes catalytic activity [64]

This work was supported by the Fondecyt Program of the Chilean National Agency for Research and Development (ANID) (Fondecyt Regular 1211384 and 1251409 to L.F.B