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RAS/MAPK activation drives resistance to smo inhibition, metastasis, and tumor evolution in Shh pathway-dependent tumors

To modify gene expression in tissues, cortisol binds to mineralocorticoid and glucocorticoid receptors 86 and additional non-genomic processes that might not be entirely dependent on certain cortisol receptors 87

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Enhancing efferocytosis to clear apoptotic cells, thereby preventing secondary necrosis and amplification of inflammation, is recognized as a central mechanism for plaque stabilization