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2017;4(1):e000155

Control of redox state and redox signaling by neural antioxidant systems

Interestingly, systemic administration of UDCA attenuates experimental auto-immune arthritis by suppressing T helper 17 cell differentiation through the upregulation of small heterodimer partner interacting leucine zipper protein and by inducing the activation of AMPK and p38 in mouse CD4+ T-cells [52]

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However, CagriBB has equivalent potency to Cagri, in CRE-reporter assays in mammalian cells expressing AMY 3 Rs (Supplementary Table 4), suggesting there is negligible impact of this rotamer difference

Additionally, there was a notable reduction in oxidative stress markers