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doi: 10.1016/j.acthis.2023.152000

The rescue experiment suggests that GSTM2 may be a useful therapeutic target for hepatic steatosis

This suggests that the pathological processes and tissue damage established over years of CFTR dysfunction may not be entirely reversible by the current levels of CFTR correction achieved, or that the restoration of CFTR function is insufficient to fully extinguish these self-perpetuating cycles of inflammation and oxidative damage

Chuang CJ

When using this stem cell source, there is an additional alloreactive mechanism that occurs involving natural killer cells

European journal of obstetrics, gynecology, and reproductive biology