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The aberrant activation of ALOX5 is important for the proliferation and migration of breast cancer cells [21], whereas the absence of ALOX5 promotes the progression of bladder cancer by enabling the evasion of ferroptosis [22]

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Future studies employing Western blot or immunohistochemistry would provide more direct confirmation of CASPASE-3 activation

Differentiation syndrome associated with enasidenib, a selective inhibitor of mutant isocitrate dehydrogenase 2: analysis of a phase 1/2 study