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These findings provide evidence of the high safety profile of this type of treatment intervention

541 A deeper exploration of mito-nuclear communication is essential for understanding the pathogenesis and heterogeneity underlying mitochondrial diseases

Radiation-induced DNA damage is sensed by the kinases ataxia-telangiectasia mutated (ATM) and ataxia-telangiectasia and Rad3-related protein (ATR), which activate downstream proteins to initiate the DNA damage response (reviewed in Maier et al., 2016)