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Description

By genomic and metabolomics analyses of patients tumours, metastasis-derived patient-derived xenografts (PDX) and isogenic cell lines we demonstrate that a fraction of metastatic ER + BC is highly reliant on oxidative phosphorylation (OXPHOS)

Cancer gene ther 12, 90100, 10.1038/sj.cgt.7700775 (2005)

In addition, another nonenzymatic process such as ROS-induced lipid peroxidation results in a variety of RCS and respective glycation products, demonstrating an interplay between oxidative and carbonyl stresses