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Abnormal accumulation of misfolded SOD1 in affected spinal motor neurons has been established as a pathological hallmark of ALS caused by mutations in SOD1 ( SOD1 -ALS)

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We used a metabolomics approach to investigate the potential molecular mechanisms of the mental health alterations in recovered COVID-19 patients

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N, N-dimethylformamide exposure induced liver abnormal mitophagy by targeting miR-92a-1-5p-BNIP3L pathway in vivo and vitro