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[DOI] [PMC free article] [PubMed] [Google Scholar] 84.Coimbra S., Oliveira H., Reis F., Belo L., Rocha S., Quintanilha A., Figueiredo A., Teixeira F., Castro E., Rocha-Pereira P., et al

At the joint inflammatory site, abnormal modulation of mTOR signaling results in continuous feedback between stromal cells and infiltrating immune cells, leading to persistent inflammation and tissue damage, driving the pathological tissue refactoring and eventually causing organ dysfunction [4]

Frailty significantly correlated with ischaemic stroke at 6 months (p 20% serially), wall motion abnormalities, acute myocardial infarction, Takotsubo syndrome, arrhythmia, or sudden cardiac death without acute atherosclerotic coronary disease (CAD)

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Dihexa works by binding to the HGF receptor c-Met on hippocampal neurons, activating intracellular signaling pathways that increase synaptogenesis, dendritic spine density, and NMDA receptor expression