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showed evidence that Cu/Zn-superoxide dismutase (SOD, Sod1 / )-deficient mice showed muscle atrophy, weakness, and neuromuscular junction (NMJ) degeneration in early adulthood, similar to normal muscle aging, and the transgenic expression of SOD1 specifically prevented muscle mitochondrial function and calcium treatment defects in neurons of SOD1-deficient mice [53]

9 Conclusion AD is a neurodegenerative disorder characterized by neuronal loss and cognitive dysfunction, featuring hallmark pathological lesions including A plaques and NFTs resulting from aberrant tau hyperphosphorylation

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(2) RS: Regulation of inflammation via microbiota-metabolite axis RS, such as RS3 derived from cooled potatoes, differs from NSPs in terms of fermentation rate and anti-inflammatory targets, demonstrating particularly pronounced effects on colonic mucosal inflammation