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While type 2 inflammation is well-characterized, many patients, particularly those with severe or neutrophilic phenotypes, respond poorly to current therapies, necessitating deeper insights into alternative pathogenic mechanisms ( - ), and lipid metabolism enzymes ( The immune system orchestrates inflammatory cascades in asthma, involving macrophages, neutrophils, T cells, and eosinophils

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To view a copy of this licence, visit About this article Cite this article Kieliszek, M., Bano, I