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NEC etiology is considered multifactorial, a consequence of intestinal immaturity, microbial dysbiosis, and an exuberant inflammatory response

have provided evidence that neuroglial cells can significantly augment neuronal energy metabolism through the coordinated utilization of FAs and KBs, especially under conditions of glycolytic impairment (McMullen et al

There is also the case of hydroxyalkyl mercapturic acids (HAMAs), which derive from GSH conjugation to several classes of carcinogens such as ethylene oxide and acrolein

The disturbances in the intestinal pathogens result in variations in the secretion of vital organs

HBx-induced HSPA8 stimulates HBV replication and suppresses ferroptosis to support liver cancer progression

Bousalis D, McCrary MW, Vaughn N, Hlavac N, Evering A, Kolli S, et al