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Aoi W, Ogaya Y, Takami M, Konishi T, Sauchi Y, Park EY, Wada S, Sato K, Higashi A

This senolytic approach, addressing DNA damage-induced senescence, offers a promising strategy for eradicating residual disease and improving cancer treatment outcomes

It works in mitochondria to help generate new cells, maintain cell functioning, and repair DNA responsible for maintaining a healthy metabolism

doi: 10.3389/fphar.2025.1648395 Received 17 June 2025 Accepted 08 September 2025 Published 03 October 2025 Volume 16 - 2025 Edited by Nunziatina De Tommasi, University of Salerno, Italy Reviewed by Sohail Mumtaz, Gachon University, Republic of Korea Lei Hu, Lushan Botanical Garden (CAS), China Updates Copyright 2025 Dubey, Chandragiri, Geevarghese, Kapoor, Gulati, Rani, Singh, Chavda, Gundamaraju, Bansal, Gautam, Goyal, Okoh, Shen and Singla

Central to this approach are degrons, short amino acid motifs, typically 610 residues in length, that mark proteins for recognition by E3 ubiquitin ligases

Liu, X.X