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Abstract Maternal cigarette smoke exposure (SE) causes detrimental changes associated with the development of chronic neurological diseases in the offspring as a result of oxidative mitochondrial damage

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Crucially, Crat knockout experiments partially reversed these metabolic improvements, establishing CrAT-mediated SREBP1c inhibition as a key mechanism through which L-carnitine counteracts de novo lipogenesis

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ade Koullar: Her trl iadeler mutlak suretle orjinal kutu ve rnn ambalaj (koruma band) bozulmakszn yaplmaldr

In addition, Trkmen et al