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The positive role of glutathione in preventing hypertension may also be explained by the observations that high glutathione levels prevent platelet-derived growth factor (PDGF)-mediated production of ROS by NADH/NADPH oxidase, which is considered to be the main source of vascular ROS (7), (22)

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This effect is reversed upon Atg7 knockout, and further amplified by concurrent Atg7 knockdown in both AML and stromal cells

However, prolonged oxidative stress and inflammation impair the functions of these cells, leading to insufficient ECM deposition, angiogenesis disorders, delayed epithelialization, and other issues that affect wound healing [45,46,47]