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Clinical trials predominantly document mild and self-limiting adverse effects such as abdominal distension, lower limb hypoesthesia, and transient pain exacerbation, which typically resolve without necessitating extensive medical intervention [4, 21, 25, 26, 66]

10.1001/jama.2011.360 JAMA

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481 This suggests that ACSL4 initiates a cascade that links ferroptosis to pyroptosis, thereby promoting cardiac hypertrophy and responding to the hemodynamic stress induced by aortic constriction

It's noteworthy that these Parkinson-like events following ranibizumab administration are distinguished by the absence of cortical and subcortical atrophy, suggesting a unique pathophysiological mechanism distinct from typical PD presentations (Sultana and Scondotto 2020)