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& Tavares, H

107 Similarly, ER stress, a hallmark of metabolic diseases, places additional demand on the UPS through the activation of the UPR

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Herein, we provide evidence that hepatic GSSG accumulation, which was observed in a high-fat diet (HFD)-fed model of NAFLD, rendered hepatocytes susceptible to TNF-induced cell death

Bjorklund et al., 2018

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