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reported that vigorous exercise in human subjects increases glutamine levels within the cortex, an effect that occurs as a consequence of activity-dependent increases in the glutamateglutamine turnover (Reference Maddock, Casazza and Buonocore66)

2,28-30 Genetic perturbation of these enzymes can, therefore, block cell death under ferroptosis-inducing conditions by eliminating the key substrate (i.e., PUFA-containing lipids) required for the execution of this process

Users of research peptides must often rely on general stability principles rather than specific data for their compounds

Chronic inhibition of mammalian target of rapamycin by rapamycin modulates cognitive and non-cognitive components of behavior throughout lifespan in mice

We first observed that SODG93A mice presymptomatic motor neurons display alterations in the coupling efficiency of oxidative phosphorylation, along with fragmentation of the mitochondrial network