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10.1038/sj.bjp.0703067

The phenotypic hallmark of the SOD1-dependent ALS is the presence of amyloid aggregates of SOD1 in affected tissues, which are the ultimate outcome of SOD1 misfolding the product of a gradual destabilization of the SOD1 structure due to the loss of metal ions and the reduction of the stabilizing intrasubunit disulfide bond 10,11,12

10.1093/nar/gkn072 96 PeltierJ

Nonsteroidal anti-inflammatory drugs (NSAIDs) such as high-dose aspirin or indomethacin have been reported to alter GH release in some contexts, though this is more pertinent to GH stimulation testing than chronic therapy

Retinoids work by overriding normal cellular behavior forcing faster turnover, suppressing oil production, chemically instructing cells to produce more collagen

The human TNF- ELISA kit (EK-H12145), human IL-6 ELISA kit (EK-H10352), and human IL-1 ELISA kit (EK-H10327) were obtained from EK Bioscience (Shanghai, China)