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Previous work has established that any residual OCR detected after oligomycin A treatment is due to non-respiratory oxygen consumption, which is usually ascribed to proton leakage through the mitochondrial inner membrane 79

Furthermore, angiopoietin-2-CMNP treatment suppressed tumor angiogenesis and activated apoptosis in cancer cells in vivo by increasing the Bax/Bcl-2 ratio and cleaved caspase-3 expression [201]