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A notable feature of FMO3, the dominant liver form, is a polymorphism (a 551C-T mutation in exon 4) that changes a proline residue (153) to a leucine, resulting in an inactive enzyme

This milestone expands the visibility and reach of the journal's research and perspectives on healthcare delivery, quality improvement, and system performance

Soluble receptor for advanced glycation end products and the risk for incident heart failure: the atherosclerosis risk in communities study

Unresolved GUD could lead to GIT perforations, bleeding, and gastric cancer (Ahmed 2020)

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J Biol Chem 271(11):60006009