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This allows the removal of damaged organelles (such as mitochondria through mitophagy, see subsection Changes in mitochondrial morphology in response to hypoxia) and release of nutrients for ongoing cell viability 175 , that promote cell survival in stressful environments (Figure 5)

Primary cultured rat tenocytes were treated with dexamethasone (100 nM, 24 h pre-treatment +24 h treatment), pefloxacin (100 M, 24 h), or both (Figure 6A)

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10.1515/revneuro-2018-0008 Rev

In the present review, we will focus mainly on the signaling cascade of TLR4 because it is believed to be the main sensor of bacterial LPS (Li et al., 2013)