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Genetic modeling demonstrated functional interactions between oxidative stress and ferroptosis genes in DC, suggesting that they may act as bridges in the pathogenesis of DC (76)

Data represent the mean SEM of one representative experiment with 3 independent replicates

instead, they found a notable decrease in the expression of cell cycle regulators such as Cyclin A1 (CCNA1) and CDK1

With the concentration of AO increased, the fluorescence intensity proportionately enhanced

After the solution had entered the column, 550 l of 100 mM HEPES p H 7.0 were loaded on the column and 300 l of 100 mM HEPES p H 7.0 were used to elute the protein

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