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Furthermore, we found that succinicGSH is secreted by FH-deficient cells and can be degraded, in a process similar to GSH catabolism, into succinic cysgly

Hence, by targeting the enzymatic activity of SARM1, inhibitors may effectively prevent the depletion of NAD + , thereby maintaining cellular energy homeostasis and enhancing neuronal resilience, potentially expanding the treatment landscape for various neurological conditions

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doi:10.1016/j.smrv.2008.12.003

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