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mTOR is also a key factor inhibiting autophagy in renal tubular epithelial cells, and leucine may regulate cellular autophagy through the mTOR pathway to attenuate renal injury

To further develop therapeutic targets, we tested the efficacy of N -acetyl cysteine (NAC), an antioxidant and anti-inflammatory agent in ameliorating the extent of myocardial damage and also investigated if facilitated repair could be augmented in a rat model of left anterior descending artery ligation-induced MI

Experimental animals A total of 48 Kun-Ming strain male mice were used

Emerging research suggests broader regenerative effects, but these remain under investigation

The disease begins with endothelial cell (EC) apoptosis, which triggers a cascade of pathological responses, including endothelial-to-mesenchymal transition (EndMT), 5 the proliferation and migration of PASMCs and PAFs, leading to ECM remodeling and fibrosis to stiffen vascular and increased pulmonary vascular resistance (PVR)

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