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(as long as the causative factor for B 12 deficiency persists, therapy should be continued) Evidence from animal and human studies shows that B 12 also has neuroprotective and anti-inflammatory properties, 15 and intervention studies in humans have reported beneficial effects of better B 12 status or B 12 therapy in multiple sclerosis, 16 Parkinsons disease, 17 myalgic encephalomyelitis, 18 autism, 19 and (administering very high doses of methylcobalamin) in amyotrophic lateral sclerosis

[26] DNA methylation is involved in the functioning of genes and is an essential epigenetic control mechanism in mammals

Without adequate B12, homocysteine levels rise, and elevated homocysteine is neurotoxic, potentially causing direct damage to nerve cells and blood vessels that supply nerves

Small amounts of the vitamin are also available in eggs, cheese, yogurt, and fortified cereal products

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Yet, that is what happens when a person with symptomatic B12 deficiency experiences that she or he needs more frequent supplementation than once every 3 months