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BPC 157 rescued NSAID-cytotoxicity via stabilizing intestinal permeability

The spleen sections revealed normal splenic architecture across all groups

Signal translation mediated by redox reactions occurs primarily by oxidation and reduction of cysteine residues

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In the xenograft model, STK38 loss or the introduction of the non-phosphorylatable RIPK1 S309A mutation rendered marked resistance to pro-necroptotic stimuli, leading to increased tumour burden (Fig