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Donati B, Vanni E, Dongiovanni P, Iavarone M, Rametta R, Rosso C, Carnelutti A, Petta S, Fracanzani AL, Reeves HL, Dofour JF, Miele L, Anstee Q, Bugianesi E, Soardo G, Fargion S, Valenti L

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These reductions may reflect a physiological adaptation to lower glucose availability and energy expenditure, rather than overt dysfunction, as the KD can enhance mitochondrial efficiency and reduce the need for high T3-driven metabolic output [48,117] (Figure 3)

A summary of these therapeutic approaches is provided in Table 2