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The synthesis of trimethylamine N-oxide (TMAO) and the development of cardiovascular risk exhibit another dimension to this dynamic activity, i.e., the interplay between the intestinal microbiome and the human host that occurs through the interaction of dietary intake (a type of environmental exposure) with the intestinal microbiota, leading to the production of metabolites that may serve as cardiac disease boosters (Tang et al., 2014a

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FPP) and these molecules also regulate HMGR at the translational level, although the molecular mechanisms behind this phenomenon are not clear [59, 6467]