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Given this critical role of GH in adjusting metabolism to the fasting condition, it is plausible that the metabolic effects of GH are required for optimal linear growth of the human fetus and that this explains marginal effects on linear growth of the human fetus

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272,273 Consistent with this, a membrane-permeating peptide that inhibits MLC kinase (PIK) was reported to prevent enteropathogenic Escherichia coli (EPEC)-, TNF-, and IFN--induced TER defects and suppress MLC phosphorylation in Caco-2 cell monolayers