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TSCI pathogenesis is characterized by two distinct phases: the primary injury, which results in irreversible mechanical damage, and the secondary injury, driven by cellular membrane disruption, apoptotic signaling, and ischemia caused by microvascular compromise [4, 5]

It is not intended to diagnose, treat or cure immune related conditions

Metaanalyses have identified specific microbial dysbiosis in PD patients, including decreased abundance of shortchain fatty acid (SCFA)producing genera (e.g., Prevotella ) and increased abundance of proinflammatory Enterobacteriaceae [121]