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Malat1 activates autophagy and promotes cell proliferation by sponging miR-101 and upregulating STMN1, RAB5A and ATG4D expression in glioma

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Moreover, tetrahydrobiopterin (BH 4 ), a crucial eNOS cofactor, is oxidized to dihydrobiopterin (BH 2 ) under oxidative stress, leading to eNOS coupling [131]