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the abnormal activation of this pathway can lead to uncontrolled cell growth and malignant transformation [15, 16], which is prominent in the development of gastric cancer associated with H

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IF: Immunofluorescence

Additionally, the authors acknowledge the general positive bias of the published results, the lack of replicative validation studies, and the resulting risk of overestimating treatment efficacy in preclinical models that would provide confidence for the current findings and improve translational significance

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These results suggest that HMOX2 localizes to both the ER and MERCs, where it interacts with MITOL