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On the basis of experimental in vitro studies, it has been strongly suggested that the inhibitory effect of iodine excess on NIS expression involves the generation of ROS

In contrast, GSH treatment did not significantly improve the GSH/GSSG ratio at any of the tested doses, indicating that exogenous GSH alone may be insufficient to restore redox balance under the given experimental conditions

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Toxicology of Glutathionine S-transferses

Multiple glutathione disulfide removal pathways mediate cytosolic redox homeostasis

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