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Treatment with inhibitor ferrostatin-1 (Fer-1) effectively suppresses high hydrostatic pressure-induced VSMC inflammation, as evidenced by reduced CXCL2 expression, and prevented endothelial dysfunction-associated phenotype switching marked by decreased AKR1C2 expression

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Providing support for a causal link between a negative energy state and increased Parasutterella abundance, a rat model of obesity, which showed similar Parasutterella relative abundances in animals maintained on HFD and those maintained on normal chow but exhibited a treatment-induced increase in Parasutterella relative abundance, displayed a negative correlation between Parasutterella and body weight 82

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Furthermore, a double mutant lacking the operon encompassing both ahpD and spr0371 genes demonstrated enhanced resistance to H 2 O 2 concentrations of 9, 18, and 37 mM in the R6 strain [29]

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