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PEG-MGF acts more locally on satellite-cell activation at the site of injection or injury and does not produce the same systemic IGF-1R signaling

[PMID: 11164475] 15

3 Initially, GSH depletion was considered as a by-product of ROS production during mitochondrial permeability during apoptosis via the intrinsic route

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The researchers hypothesized that thiol-disulfide exchange reactions with DSF decreased the bioavailability of bacillithiol and cysteine to thwart the ability of fosB to neutralize fosfomycin

doi: 10.3390/metabo12121247