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ATF6 as a transcription activator of the endoplasmic reticulum stress element: thapsigargin stress-induced changes and synergistic interactions with NF-y and YY1

For example, several studies have linked the occurrence of excess protein and aggregated protein deposition with diseases

Some research suggests it may help maintain telomere length, which is associated with aging

Additionally, its anti-inflammatory effects create a less favorable environment for bacterial proliferation

Moreover, S100A2 interacts with p53 to increase its transcriptional activity, and posttranslational modification of p53 increases its interaction with S100A2 (58)

Connection between systemic inflammation and neuroinflammation underlying neuroprotective mechanism of various phytochemicals in neurodegenerative diseases