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The aforementioned observations prompt the question of whether NF-B directly regulates EAAT3 expression at the transcriptional level

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However, recent genomic, proteomic and functionomic advances have unveiled unexpected disease-associated mutations and altered expression, secretion and interactions in human ARSs, revealing hidden biological functions beyond their catalytic roles in protein synthesis

These molecular events ultimately contribute to an increase in the number and functionality of synapses, leading to amelioration of depressive symptoms

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