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to human FcRn-expressing mice together with CpG adjuvant, followed by a 10% booster dose after 3 weeks

Functional data on Dnm2 reveals the possible pathophysiological mechanisms via which Dnm2 mutations can lead to two distinct neuromuscular disorders

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Mild redness and swelling may be possible

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Possible heterogeneity may also mean that one single biological measure may not be feasible as a marker for ME/CFS