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Promotion of adipogenesis by 15-(S)-hydroxyeicosatetraenoic acid

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Sakamoto, T., Maebayashi, K., Nakagawa, Y

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Five common variants in four GST genes ( GSTM1, GSTT1, GSTP1, and GSTM3, described in Table 1) have been studied extensively in relation to lung cancer, with each associated with completely lost or reduced activities of certain xenobiotic metabolizing enzymes: (i) the GSTM1 *0 ( GSTM1 null), (ii) the GSTT1 *0 ( GSTT1 null) alleles represent deletions of the GSTM1 and GSTT1 genes, respectively, with each conferring a total loss of activity in their corresponding enzymes [5], (iii) the A to G transition in GSTP1 that gives rise to the Ile105Val polymorphism (also known as I105V), (iv) the C to T exchange at position 341 in the same gene, which results in the Ala114Val polymorphism (also known as A114V) (both of these GSTP1 polymorphisms confer moderately reduced enzyme activity) [143], and (v) the GSTM3 intron 6 polymorphism, a 3-base pair deletion in intron 6, which is in linkage disequilibrium with the GSTM1 genotype and contains a recognition motif for the YY1 transcription factor, which has been postulated to regulate gene expression [144146]

Oxidoreductases are key enzymes in central carbon metabolism that catalyze oxidationreduction reactions 11,12