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3 Mechanisms underlying cell death in cardiovascular disease Cell death is a central pathological event in CVDs, contributing to the progression of conditions such as atherosclerosis, myocardial infarction, and heart failure (Figure 2) ( Figure 2 3.1 Programmed cell death in cardiovascular diseases PCD refers to tightly regulated cellular processes involving molecular pathways that orchestrate cell demise ( Apoptosis, a caspase-mediated process, is pivotal in both acute and chronic cardiovascular injury ( Autophagy is primarily a survival mechanism, but excessive autophagy contributes to cell death under stress conditions, such as hypoxia or oxidative stress ( Iron-dependent lipid peroxidation characterizes ferroptosis, which is distinct from other PCD modalities (102)

Botta T, Blescia S, Martnez-Heredia J et al (2009) Identificacin de diferencias protemicas en muestras oligozoosprmicas

Altering ethanol pharmacokinetics to treat alcohol use disorder: can you teach an old dog new tricks

Mitochondrial injury directly impairs the function of alveolar epithelial cells, thereby triggering local inflammatory responses and promoting fibroblast activation driven by TGF-, a process that plays a critical role in the development of pulmonary fibrosis (Liu and Chen, 2017

Glutathione is a naturally occurring antioxidant made in the body from amino acids such as cysteine, glycine, and glutamic acid

PMID: 30342045