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We observed increased TF activity for AP-1 family members (e.g., JUN and FOS) as well as other TFs identified through differential footprint analysis in the bulk stage (e.g., NFKB1, CREB1, ATF2, EGR1) with continuous exposure to ART ( Figure 8C )

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Many are confined to a re-discussion of Nrf2 and HDAC inhibition, lacking integration with its biosynthetic origins and failing to synthesize the rapidly expanding body of evidence on novel and underappreciated mechanisms

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MITOL knockout in both HeLa and C2C12 cells resulted in reduced mitochondrial iron levels (Extended Data Fig