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Further, the study demonstrated reduced levels of ROS and enhanced activities of superoxide dismutase, which have been linked to cellular survival, leading to many pathological conditions such as cancer

The crosstalk of both may serve as a central hub for drug-induced toxic responses

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The release of free copper ions, facilitated by transporters such as ATP7A and ATOX1, induces neointimal thickening following vascular injury, contributing directly to lesion development [193, 194]