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Guo et al., 2022)

Specifically, the study highlighted the ability of TB-500 to enhance cardiac function in response to oxygen deprivation [9]

They proposed that mTORC2/SGK1 signaling regulates mitochondrial homeostasis, and that induction of autophagy in mTORC2/SGK-1-deficient animals is triggered by mitochondria-derived reactive oxygen species (mtROS) [59]

Beside its role as reactive oxygen specie and therefore, causer of oxidative stress, the H 2 O 2 surplus causes the hemoglobin oxidation and, thus, diminution of the oxygen concentrations, what can cause infections, ulcer and even necrosis (Wieacker et al, 1980)

10.1016/j.gene.2018.06.058 Gene 172 LightowlersR

Contrarily, a study with 8 GH-deficient subjects found that IGF-1 administration increased lipid oxidation (being this effect more potent when co-administered with GH), energy expenditure, and insulin resistance [108, 183]