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The ability of quercetin to suppress the production of pro-inflammatory cytokines is mediated by its suppression of the activation of ERK and p38 MAP kinase and NF-B/IB signal transduction pathways (Tang et al., 2019)

doi: 10.1002/mds.29056 78 KnopmanD

Importantly, damage-associated molecular patterns (such as mitochondrial DNA, fibronectin extra domain A, and galectin-3) released during tissue damage may activate NETs through interaction with neutrophil surface proteins [172]

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